Oral Presentations
2019

• Bhatnagar, S. From genetics and bioinformatics to function: identification and characterization of novel protein inhibitors of insulin secretion.  Department of Cell, Developmental and Integrative Biology, University of Alabama, Birmingham, Al. November 20, 2019.
  
• Bhatnagar, S. From genetics and bioinformatics to function: identification and characterization of novel protein inhibitors of insulin secretion. Department of Medicine, Division of Endocrinology, Diabetes, and Metabolism, University of Pittsburgh, PA. November 13, 2019.
  
• Bhatnagar, S. Identification and characterization of complement 1q-like 3 secreted protein in β-cell function, Endocrinology Research Meeting, UAB, Birmingham, AL, August 13th, 2019. 
• Bhatnagar, S. Regulation of insulin secretion from pancreatic beta cells. Endocrinology Grand Rounds, UAB, Birmingham, AL, February 28th, 2019.
• Gupta, R and Bhatnagar, S. Complement 1q like-3 Secreted Protein Inhibits Insulin Secretion by an Adhesion G-Protein Coupled Receptor, BAI3 in Pancreatic β-cells. Midwest Islet Club Conference, Ann Harbor, MI, May, 2019.
• Gupta, R and Bhatnagar, S. Complement 1q like-3 Secreted Protein Inhibits Insulin Secretion by an Adhesion G-Protein Coupled Receptor, BAI3 in Pancreatic β-cells. University of Alabama Diabetes Research Day, Birmingham, AL, May, 2019. Top scoring abstract-Winner of the 2019 Eliezer S. Award for Best Diabetes Research​

2018

• Gupta, R and Bhatnagar, S. An alpha-cell secreted protein, complement 1q like-3 inhibits insulin secretion via BAI3 adhesion G-protein coupled receptor from pancreatic beta-cells. Midwest Islet Club Conference, St. Louis, Missouri, May, 2018.  ​
• Bhatnagar, S. From genetics and bioinformatics to function: identification and characterization of novel protein inhibitors of insulin secretion. Seminar at Dept. of Physiology, Johns Hopkins University.
• Bhatnagar, S.  The complement 1q-like 3 secreted protein acts as an inhibitor of insulin secretion from pancreatic β cells. Keystone Symposia on the Frontiers of Islet Biology, Keystone, CO.
• ​Huynh, T.C., Truong, C., Gupta, R., Schueler, K., Attie, A.D., and Bhatnagar, S. Synaptotagmin-9 regulates tomosyn-2 protein abundance to affect early phase of insulin secretion. Boshell Diabetes Research Day, Auburn, AL.
• Gupta, R.,Koltes, J.E., and Bhatnagar, S. The complement 1q-like 3 secreted protein inhibits insulin secretion by G-protein coupled receptor, BAI3 from pancreatic β-cells. Boshell Diabetes Research Day, Auburn, AL.
• Huynh TC, Gupta R, Bhatnagar, S. Regulatory mechanism of the early phase of insulin secretion by tomosyn. UA Systems Honors Research Day, Tuscaloosa, AL.

2017

• Huynh, T.C., Gupta, R., Bhatnagar, S. Synaptotagmin-9 regulates tomosyn protein abundance to affect early phase of insulin secretion. UAB Fall Research Expo, Birmingham, AL. 
• Bhatnagar, S.  C1ql3 antagonizes GLP-1-induced insulin secretion by Bai3 adhesion G-protein coupled receptor in pancreatic beta cells. American Diabetes Association Conference: 77ndScientific Sessions, San Diego, CA. 
• Huynh, T.C., Truong, C., Gupta, R., Schueler, K., Attie, A. D., and Bhatnagar, S. Synaptotagmin 9 Regulates Tomosyn-2 Protein Abundance to Affect Early Phase of Insulin Secretion. Midwest Islet Club Conference, Madison, WI.
• Gupta, R., Koltes, J.E., and Bhatnagar, S. C1ql3 Antagonizes GLP-1 Induced Insulin Secretion by Bai3 Adhesion G-Protein Coupled Receptor in Pancreatic Beta Cells. Midwest Islet Club Conference, Madison, WI.
• Gupta, R., and Bhatnagar, S. C1ql3 Antagonizes GLP-1 Induced Insulin Secretion by Bai3 Adhesion G-Protein Coupled Receptor in Pancreatic Beta Cells. UAB Diabetes Research Day, Birmingham, AL.
• Gupta, R., Huynh, T.C., Truong, C., Nguyen, D., and Bhatnagar, S. An Islet Secreted Protein, C1ql3 Inhibits GLP-1 Induced Insulin Secretion via Bai3 Adhesion G-protein Coupled Receptor from Beta Cells. Boshell Diabetes and Metabolic 10th Annual Research Day, Auburn University, AL.

2016

• Bhatnagar, S. Phosphorylation and degradation of tomosyn-2 de-represses insulin secretion Boshell Diabetes and Metabolic 9th Annual Research Day, Auburn University, AL.

2015

• Bhatnagar, S. Phosphorylation and degradation of tomosyn-2 de-represses insulin secretion Midwest Islet Conference, Chicago,IL.
• Bhatnagar, S. Identifying and characterizing novel regulators of insulin secretion. Seminar at the Department of Molecular Medicine, University of South Florida, Tampa, FL.

2014

• Bhatnagar, S. Identifying and characterizing novel regulators of insulin secretion. Seminar at the Obesity and Diabetes Center, University of Louisville, Louisville, KY.
• Bhatnagar, S. Identifying and characterizing novel regulators of insulin secretion. Seminar at the Comprehensive Diabetes Center, University of Alabama at Birmingham, Birmingham, AL.

2013

• Bhatnagar, S. Identifying and characterizing novel regulators of insulin secretion. Spring Seminar Series, Department of Biochemistry, West Virginia University, Morgantown, WV.

2012

• Bhatnagar, S. Positional Cloning of a Type 2 Diabetes Quantitative Trait Locus: Tomosyn-2, a Negative Regulator of Insulin Secretion. Membrane Protein/Trafficking Seminar Series. Bock Laboratories, University of Wisconsin-Madison, WI.

2011

• Bhatnagar, S. Positional cloning of a type 2 diabetes quantitative trait locus; Tomosyn-2, a negative regulator of insulin secretion. The Midwest Islet Club Conference, University of Wisconsin-Madison, Madison, WI.

2008

• Bhatnagar, S. Fibroblast Growth Factor-19: A Novel Factor for Inhibiting Fatty Acid Synthesis. E.J. Van Liere Research Convocation and Research Day, West Virginia University, WV.

2005

• Bhatnagar, S. New Insights in Eukaryotic Transcription Termination. Department of Biochemistry and Molecular Pharmacology, West Virginia University, WV.